Human Complement Factor H, CFH ELISA Kit, 5*96T Shaker&Mixing Mutations in MYL3 have been

Mutations in MYL3 have been identified as a cause of mid-left ventricular chamber type hypertrophic cardiomyopathy

this protein is coupled to a transient receptor potential (TRP) ion channel through a G protein signaling pathway and produces a physiologic light response via membrane depolarization and increased intracellular calcium

Can promote transcription activation through binding to APBB1-KAT5 and inhibit Notch signaling through interaction with Numb (By similarity)

The protein encoded by this gene is cell-cycle regulated| and has nuclear localization

Human Complement Factor H, CFH ELISA Kit, 5*96T Shaker&Mixing Mutations in MYL3 have been